HRIDROGA SAMPRAPTI

• DEFINITION:-
)id baaQaaM p`kuva-int )d`aogaM
tM p`caxato l
Impairment of functions of heart
is Hridroga.
• ]%qaana- AamaaSayasamau%qa
• daoYa- i~daoYa
• dUYya- rsa, r>, maaMsa, maod,
Aaoja
• sa`aotaoduYTIp`kar- saMga,
Aitp`vaRi<a, isaraga`Miqa
• maaga-- maQyama
• p`sar- rsavah, r>vah, Annavah,
manaaovah s~aotsa
• sqaanasaMEaya- )dya
• svaBaava- AaSaukarI, icarakarI
 CLASSIFICATION OF
HRIDROGA SAMPRAPTI
1. ACCORDING TO HETU
2. ACCORDING TO SHATKRIYA
KALA
3. SAMANYA SAMPRAPTI
4. ACCORDING TO AYURVEDIKA
GRANTHA
 ACCORDING TO HETU

• IT IS CLASSIFIED INTO THRRE

TYPES
2. MANASIKA- AHARAJA AND
VIHARAJA
3. SHARIRIKA
4. AGHATAJA
SAMPRAPTI ACCORDING TO
MANASIKA HETU

• HETU- CHINTA, BHAYA, KRODHA,

SHOKA, ETC.
• CHITNA AND ATIDHYANA-
AGGRAVATION OF PRANA AND
VYANA VAYU
 CHINTA

AJIRNA

VITIATION OF PACHAKA AND SADHAKA PITTA

VITIATION OF KLEDAKA AND AVALAMBAKA KAPHA

(HRIDAYA IS SITE FOR KAPHA)

SO IT CAUSES IMPAIRMENT IN FUNCTIONS OF HEART

IMPAIRMENT IN HEART AND RASA- RAKTA VIKSHEPANA

DHVANI VIKRUTI AND OTHER IMPAIRMENT OF HEART

• INVOLVED DHATUS, RASA, RAKTA,
MANSA AND MEDA
• DHATUS WILL VITIATE
UAPDHATUS
• MAINLY SIRA AND SNAYU ARE
INVOLVED
• ASHTABINDU OJA OR APARA OJA
IS SITUATED IN HRIDAYA
SAMPRAPTI ACCORDING TO
SHARIRIKA HETU
• DUE TO AHARA-
• HETU- ATIRUKSHA, SHUSHKA,
USHNA, GURU, SNIGDHA, KATU,
LAVANA, AMLA RASATMAKA,
KSHARIYA, MADHYATIREKA,
ATYALPA BHOJAN.
• KAPHA MEDA SANCHAYA IN THE
LUMEN OF CORONARY VESSELS
(STROTORODHA).
DEPOSITION OF KAPHA AND MEDA
IN LUMEN OF CORONARY VESSELS.
↓
LESS BLOOD SUPPLY TO MYOCYTES
↓
IHD DUE TO ATHEROSCLEROSIS
(DHAMANIPRATICHAIA)
 DHATU KSHAYAJANYA AHARA
↓
IMPROPER NOURISHMENT OF DHATU
WHICH ARE RELATED WITH HRIDAYA
↓
IMPAIRMENT IN FUNCTION FOF HEART
↓
HRIDROGA
DUE TO VIHARA-

VEGA VIDHARANA (SUPPRESSION OF URGE OF MUTRA AND PURISH)

APANA VAYU VIKRUTI

ACCUMULATION OF MALA IN THE

BODY

UPWARD MOVEMENT OF
APAN VAYU

VITIATION OF PRANA AND VYANA VAYU

IMPROPER RASA RAKTA VIKSHEPANA

DISTURBED NUTRITION

DIFFERENT KINDS OF HEART DISEASES

SAMPRAPTI ACCORDING
TO AGHATAJA HETU
• HETU-KRIMI SANKRAMANA,
AMAVATAJANYA HRIDROGA,
CO-AGULATION CASCADE
SAMPRAPTI ACCORDING TO
SHATKRIYAKALA
• HETU- IMPROPER AHAR, VIHARA,
INSTABLE MENTAL STATUS
• SANCHAYA-ACCUMULATION OF
KAPHA IN ITS SITE OF
PREDOMINANCE I.E. CHEST
REGION.
• SANCHAYA-INCREASE OF KAPHA
IN CHEST REGION .
INCREASED DOSHAS WILL
FURTHER AGGRAVATE.
• PRASARA-THEY FLOW OUT OF
THE SITE AND SPREAD ALL OVER
THE BODY.
VITIATES RASAVAHA AND
PRANAVAHA STROTASA
• STHANASANSHRAYA-INCREASED
DOSHAS LODGE INTO VARIOUS
PARTS OF THE BODY LIKE
CORONARY ARTERIES, VALVES OF
THE HEART, MUSCLES OF THE
HEART.
• VYAKTI-LEADING TO CORONARY
ARTERY DISEASE (CHD), VALVAR
ABNORMALITY,
MYOCARDIATIS,ETC.
• BHEDA- IF LEFT UNTREATED
THEN LEADS TO COMPLETE
MANIFESTATION RESULTING IN
DIFFERENT KINDS OF
HRIDROGAS E.G.- HRUTSHULA,
HRUD-DRAVA, ETC.
SAMANYA SAMPRAPTI OF
HRIDROGA
• dUYaiya%vaa rsaM daoYaa
ivagauNaa )dM gata: l
• )id baaQaaM p`kuva-int )d`aogaM
tM p`caxato l l
(sauEaut ]<ar
KMD)
 VITIATION OF
PRINCIPLESOF DIET
AND OTHER NIDANA

MANDAGNI

AMOTPATTI

SAAMA RASA

VITIATED RASA+ KAPHA +

VITIATED RASA+VAYU
PITTA

RUJA IN HEART STROTORODHA

OBSTUCTION OF PRANA VAYU

HRIDA BADHA
 SAMPRAPTI ACCORDING TO
ACCORDING TO AYURVEDIC
GRANTHA
• ASHTANG HRIDAYA AND MADHAVA
NIDANA HAS BEEN DESCRIBED FIVE
TYPES OF HRIDROGAS.
• THESE ARE NAMELY-
3. VATAJA
4. PITTAJA
5. KAPHAJA
6. SANNIPATAJA AND
7. KRIMIJA.
 VATAJA HRIDROGA

• HETU- SHOKA, UPAVASA,

VYAYAMA, RUKSHA, SHUSHKA,
ALPA AND SHITA AHAR.
• RESULTS IN VATA PRAKOPA AT
THE SITE OF HRIDAYA BY
STHANASANSHRAYA PRODUCE
HRIDROGA
 PITTAJA HRIDROGA
• HETU-ATIUSHNA, AMLA, LAVANA,
KSHARA, KATU RASATMAKA AHAR
ALSO EXCESSIVE CONSUMPTION OF
FOOD, AJIRNA BHOJANA,
MADHYAPANA, KRODHA,
ATAPASEVANA, ETC.
• CAUSE PITTA PRAKOPA AT THE SITE
OF HRIDAYA LEADING TO HRIDROGA.
 KAPHAJA HRIDROGA
• SHOWS THE SYMPTOMS LIKE
HEAVINESS AT THE SITE OF
HEART, STAIMITYA, KASA,
TANDRA, JWARA, ANOREXIA,
EXCESSIVE KAPHASHTIVANA
 SANNIPATAJA
HRIDROGA
• SHOWS THE SYMPTOMS OF
ABOVE THREE TYPES.
• THE MAIN SYMPTOMS ARE
VAIVARNYA, SHVASA, JWARA,
SHULA, SHOTHA, HRID-
DHVANIVIKRUTI.
KRIMIJA HRIDROGA
SUFFERING FROM TRIDOSHAJA HRIDROGA

CONSUMPTION OF TILA, DUGDHA, GUDA, ETC KAPHAKAR AHAR

GRANTHI UTPATTI IN THE HRIDAYA

THAT PART BECOMES VIKRUTA

PRODUCTION OF KLEDA BY RASA IN THAT VIKRUTA PART

KRIMI UTPATTI FROM KLEDA

HRIDROGA
COMMON SYMPTOMS OF
HRIDROGA
• vaOvaNya-maUcCa-jvarkasaih@ka
XvaasaasyavaOrsyatRYNaa
p`maaoha: l
• Cid-: kfaot\ @laoSa$jaao|$icaXca
)d`aogajaa: syauiva-
ivaQaastqaa\nyao ll
(cark icaik%saa)
• Prana Vayu Vikruti- Shwasa, Kasa,
Hikka, Moha;
• Vyana Vayu Vikruti -Hritspandan and
Shotha;
• Rasa Dushti- Jwara, Trishna, Moha,
Shotha, Kaphotklesha, Aruchi,
Chardi; Rakta Dushti- Vaivarnya;
• Oja Dushti- Vaivarnya, Moha,
Murccha and
• Mana Dushti- Utsahahani, Fear and
Angasada
 “PATHOGENESIS OF
ISCHEMIC HEART DISEASE”
• DEFINITION-IHD IS THE
GENERIC DESIGNATION FOR A
GROUP OF CLOSELY RELATED
SYNDROMES RESULTING FROM
MYOCARDIAL ISCHEMIA AN
IMBALANCE BETWEEN SUPPLY &
DEMAND OF THE HEART FOR
OXYGENATED BLOOD.
THE CLINICAL MANIFESTATIONS
OF IHD CAN BE DIVIDED INTO
FOUR SYNDROMES-
1. MYOCARDIAL INFARCTION (MI),
2. ANGINA PECTORIS,
3. CHRONIC IHD WITH HEART
FAILURE AND
4. SUDDEN CARDIAC DEATH
• Although only a single major coronary
epicardial trunk may be affected,
two or all three- left anterior
descending (LAD), lateral circumflex
(LCX), and right coronary artery
(RCA) - are often involved
 ANGINA PECTORIS
• DEFINITION-ANGINA PECTORIS is a
symptom complex of IHD characterized by
paroxysmal and usually recurrent attacks
of substernal or precordial chest
discomfort (variously described as
constricting, squeezing, choking, or
knifelike) caused by transient (15 seconds
to 15 minutes) myocardial ischemia that
falls short of inducing the cellular
necrosis that defines infarction.
 There are three over
lapping patterns of angina
pectoris-
1. Stable or typical angina,
2. Prinzmetal or variant angina and
3. Unstable or crescendo angina.
Stable or typical angina,
• Stable angina, the most common form and
therefore called typical angina pectoris,
appears to be caused by the reduction of
coronary perfusion to a critical level by
chronic stenosing coronary
atherosclerosis.
• this renders the heart vulnerable to
further ischemia whenever there is
increased demand such are that produced
by physical activity, emotional, excitement
or any other cause of increased cardiac
workload.
Prinzmetal or variant angina
• Prinzmetal variant angina is an uncommon
pattern of episodic angina that occurs at
rest and is due to coronary artery spasm
usually there is an elevated ST segment on
the electrocardiogram (ECG), indicating of
transmural ischemia. Although individual
with this form angina may well have
significant coronary atherosclerosis, the
anginal attacks are unrelated to physical
activity, heart rate, or blood pressure
 Unstable or crescendo
angina
• Unstable or crescendo angina refers
to a pattern of pain that occurs
progressively increasing frequent, is
precipitated with less effort, often
occurs at rest, and tends to be of
more prolonged duration
MYOCARDIAL INFARCTION
• MYOCARDIAL INFARCTION is also
known as ‘Heart attack’, is the death of
cardiac muscles resulting from ischemia. It
is by far the most important form of IHD
and alone is the leading cause of death.
Most myocardial infarcts are transmural,
in which the ischemic necrosis involves the
full or nearly full thickness of the
ventricular wall in the distribution of a
single coronary atherosclerosis, acute
plaque changes, and superimposed
thrombosis.
• MI may occur at virtually any age.
But the frequency rises progressively
with increasing age and when
predispositions to atherosclerosis
are present, such as hypertension,
cigarette smoking, diabetes mellitus,
genetic hypercholestoremia, and
other causes of hyperlipoproteinemia.
 THE DESIGNATION
CHRONICISCHEMIC HEART
DISEASE (CIHD)
• THE DESIGNATION
CHRONICISCHEMIC HEART
DISEASE (CIHD) is used here to
describe the cardiac finding in
patient, often but not exclusively
elderly, who develop progressive
heart failure as a consequence of
ischemic myocardial damage.
 SUDDEN CARDIAL
DEATH (SCD)
• SUDDEN CARDIAL DEATH (SCD)
is most commonly defined as
unexpected death from cardiac
causes early after symptom onset
(usually within one hour) or without
onset of symptom in many adults,
SCD is a completion and often the
first clinical manifestation of IHD.
• With decreasing age of the victim, the
following nonatherosclerotic causes of
SCD become increasingly probable –
congenital structural or coronary arterial
abnormalities, Aortic valve stenosis, Mitral
valve prolapse, Myocarditis, Dilated or
hypertrophic Cardiomyopathy, Pulmonary
hypertension, Hereditary or acquired
abnormalities of the cardiac conduction
system, Isolated hypertrophy,
hypertensive or unknown causes.