FrenZzz yang baik hati, soal ini jangan di fotokopi di lingkungan FK, pokoknya usahakan jangan sampai terlihat

oleh dosen, staf FK, apalagi UPEP-ers, key! Semangat y0w Belajar! Chaiy0o!!! n_n Soal MCQ Blok 10 PDU 07 1. Bicuspid valve a. left side heart b. Guard auricle heart c. Guard entrance aorta d. Guard entrance pulmonalis 2. Freshly oxygenated blood enters the heart through _____ and pumped out the ___ left atrium, aorta. 3. Characteristic of ducting elastic artery (conducting artery) a. Tunica adventitia is composed by loose connective tissue b. Vasa vasorum is subendothelial layer c. Tunica media fenestrated by elastic membrane d. Thick internal elastica lamina (undulating) 4. General structure of Blood vessels in histological section, the arteri is round, with blood inthe lumina. 5. Struktur histologi otot jantung a. Multinucleated dan tidak bercabang b. Inti sel terletak di tepi c. Fusiform in shape, inti di tengah d. Individual cell branch and connect to cell via desmosome 6. Histology structure sel purkinje a. Branch b. Bigger cell c. Cytoplasma contain abudent d. In subendothelium layer 7. The vascular system of human embrio developed from a. Mesenchymal cell b. Angiogenetic cluster c. Cardogenic area d. Splancnic mesoderm layer 8. Bagian jantung primitif .... endocardial tube .... berubah menjadi epimiocardial mantle 9. A persons EKG dengan normal QRS n T, tidak ada P wave, pace makernya (lokasi) a. SA nodes b. AV nodes c. Bundle Hiss d. Purkinje 10. 11. Which one of the appropriate for stroke volume a. Amount by blood pumped by heart in a minute b. Amount by blood pumped by Atrium c. EDV-ESV d. ESV-EDV 1

12. The blood circulation of our body Left ventrikel Aorta Artery Capillary Vein Vein Cava Right atrium 13. In exercise, CO increased by a. BP b. HR c. SV d. SV dan HR 14. Bunyi jantung pertama a. Tricuspid dan mitral valves closed 15. EDV=120, ESV=50, HR=70 Ditanya CO 4900 16. Hipertensi bisa menyebabkan hipertrofi a. Aorta b. Left ventrikel c. Right ventrikel d. Right atirum 17. In phisiology characteristic, the cardiac muscle difference with other muscle cells that a. Cardiac muscle has striate b. Cardiac muscle work involuntary c. Cardiac muscle is fatique resistance d. Cardiac muscle has pace maker 18. Muscle stimulate aksi potensial a. Sino Atrial nodes 19. 20. Gambar AV Blok 3 21. 22. Jika pengobatan pertama tidak berhasil, maka dilakukan Pacemaker permanent 23. A 58 yo man with history 8 days palpitation and takes warfarin. BP 128 mmHg. His exam is normal. ECG shows AF with rapid ventrikuler rate. Therapy, except a. Digoxin b. propanolol 24. WPW syndrome is not characterized by which one following a. This syndrome may occur in patient with valvular abnormalities, cardiomiopathy, or otherwise normal heart function b. The arrythmias is denoted by ventrikuler preexcitation c. The arrythmia is characterized by a delay of the impulse in the AV nodes region d. The PR interval is shortened (the impulse reaches the ventrikel without going through the AV nodes) due to the activating of an alternate, shorter pathway to the miocardium 25. Conduction Velocity is most rapid in which tissue a. Purkinje-His b. Atrial c. AV nodes d. Ventricel 2

26. Repolarization of the miocardial cells is determined mostly by which current a. Outgoing sodium b. Ingoing calsium c. Outgoing potassium d. Ingoing chloride 27. 28. 29. Obat antiaritmia berdasarkan klasifikasi Vaughan Williams (Kelas I, II, III, IV). Di bawah ini temasuk obat antiaritmia tapi bukan termasuk klasifikasi adalah a. Amiodarone b. Procainamide c. Sotalol d. Adenosine 30. 31. Digoxin can be administered orally for long term suppression of atrial flutter/fibrilation. Digoxin produces therapeutic effect by a. Blocking Na+, K+ ATPase and indirectly increasing intracelluler calsium b. Centrally increasing the activity of N. Vagus c. Blocking sodium channel on the membrane of cardiac cell d. Increasing the force of cardiac contraction 32. 33. Efek samping amiodarone fibrose pulmonal 34. Obat antiarythmia, -blocker sotalol 35. Obat CCB punya fungsi antiarithmia, antihipertensi, dan antiangina a. Verapamil b. Disopyramide c. Ibutilide d. Quinidine 36. Class I drugs have antiarythmic action, while is resulting from the blockade of sodium channel on the cardiac cell membrane. Choose one from the listed drugs below, when is an example of class I antiarrythmic drugs a. Propafenone b. Diltiazem c. Propanolol d. Amiodarone 37. 38. In general, treatment of asymptomatic/ minimally symptomatic arythmias should be avoided coz ... 39. Nitrogliserin adalah obat pilihan untuk serangan angina acute, efeknya adalah a. Venodilatation b. Arteriodilatation c. Veno & arteridilatation d. Veno & arteridilatation, coronary collateral dilatasi 3

40. Nitrogliserin dikonsumsi secara sublingual karena ? 41. Verapamil is a very powerful Ca antagonist that reduce the frquency and force of cardiac contraction. Bcoz of that, verapamil should not be combinate with Propanolol 42. Beta blocker are very effective in the prophylaxis of anginal attack. The therapeutic effect are result from a. Vasodilatation b. Decrease heart rate and force of contraction c. Arterial vasodilatation d. Increased miocardial O2 consumption 43. Aspirin sebagai obat anti platelet bekerja dengan menghambat a. Protein kinase b. COX-1 c. Cytokrom p-450 d. Miosin kinase 44. Heparin is oftenly used in the treatment of acute coronary syndrome, Heparin always administered Parenterelly cause? a. Heparin is not absorbed per oral b. Heparin is destroyed in the strip c. Heparin undergoes massive effect first past d. Heparin can cause internal bleeding 45. Meta analisis uji klinik aspirin dan obat anti platelet lainnya confirm benefit intervensi prevention sekunder of vascular event among of the patient with history vascular event. Obat anti platelet lainnya termasuk a. Clopidogrel & Triclodipine b. Heparin c. Warfarin d. Vit K 46. Warfarin produce its anticoagulant effect by inhibit action of a. Cyclooxigenase 2 b. Vit K reductase c. Protein kinase A d. ACE 47. The most common morphological disorder involving valve caused by endocarditis in RHD a. Aortic insufficiency b. Aortic stenosis c. Mitral stenosis d. Trcuspid insufficiency 48. Pathologic disorder of the cor in RHD a. Endocarditis b. Miocarditis c. Pancarditis d. Pericarditis 49. Microscopically features of miokarditis in RHD are found spesific granulomatous lession dispressed in miometrium layer especially in intraventrikuler septum, the lession called a. Veruccae b. Mc callum c. Aschoff d. Tubercle 4

50. The most underlying disease in bacterial endocarditis is a. ASD b. ToF c. Mitral stenosis from RHD d. Pulmonary stenosis 51. The basic reaction of autoimmune process in rheumatic fever a. Delayed reaction hipersensitivity b. Complement mediated citotoxicity c. Cross reaction antibody in tissue antigen (Cardiac antigen) d. Reaction of hipersensitivity 52. The disorder of arterioles which is thickening the wall and plugs of hyaline mass within are found in a. Chronic mild Hypertension b. Malignant Hypertension c. Hyperlipidemia d. Acute severe Hypertension 53. If in the microscopically finding there are a lot of foam cell in subintima layer is called a. Atherosclerosis b. Fatty streaks c. Fatty plaque d. Injured intima 54. The mayor function of VLDL is a. Delivery cholesterol b. Delivery of dietary fat and other fatty acid c. Delivery of carbohidrat and protein d. Picking up (scavenging) excess cholesterol from cell to liver 55. The mayor function of HDL is a. Delivery cholesterol b. Delivery of dietary fat and other fatty acid c. Delivery of carbohidrat and protein d. Picking up (scavenging) excess cholesterol from cell to liver 56. The mayor function of cylomicron is to a. Deliver fatty acid via lipoprotein lipase b. Deliver cholesterol from liver to cell c. Endogenous trigliceride d. Membawa cholesterol dari cell ke liver 57. Chilomicrons are synthesized in a. Liver b. Adipose cell c. Coloni d. Small intestine 58. VLDL are synthesized in a. Muscle b. Adipose cell c. Small intestine d. Liver 5

59. LDL are synthesized a. From HDL b. From IDL c. In liver d. In fat cell 60. In dislipidemia, concentration of lipoprotein a. HDL meningkat b. LDL > 135 c. Total cholesterol dan TGs < 150 d. VLDL normal 61. Characteristic dislipidemia TG, HDL 62. Initial Blood preesure a. Genetic 50% b. Environment 30 % c. One locus d. Interact with other genes 63. Dilated Cardimyopathy (Tentang genetic disorder) a. 25 % genetik b. Usually X-linked c. Rare autosomal dominant d. 8 gene loci unknown 64. Faktor risiko atherosclerosis, kecuali a. Male sex b. advance age c. Hipotensi d. Insulin resistance 65. Nutrition intervention of Heart failure b. Sodium 2000 mg recommendation 66. At 1st food/fase awal recommendation for ischemic heart disease a. liquid b. semi solid c. Soft d. Solid 67. ECG for ASD RAD, RVH, RBBB 68. 69. What is natural history of small VSD? a. Polycitemia b. Spontaneous closure c. Cardiomiopathy d. CHF

70. Yang menyebabkan sianosis pada TGA a. Mixing b. Independent pulmonary-systemic flow c. Inadequate pulmonary flow d. Increased extreme pulmo 71. 72. The phisiology of tricuspid atresia a. All systemic venous blood is directed to LA b. Systemic and pulmonary circulation function in parallel c. The direction a magnitude of flow through the defect depend on pulmonal stenosis d. The pulmonary and systemic circuit may be equal 73. 74. ECG pada ToF RVH 75. In long standing hypertension with normal LV function, physiological respon the heart is a. Adaptive dilatasi b. Adaptive hypertrophy c. Dilatasi diikuti hipertrofi d. All are true 76. Cause of HF a. CAD b. Hypertension c. Valve disease d. All above 77. Which of the following is a compensation mechanism on HF a. neurohormonal system b. HR c. Na and water retention d. All are true 78. 79. The true treatment of heart failure a. Diuretics to reduce Na and water retention b. Digitalis when there is the problem of contractility c. B-blocker with caution for contraindication d. All are true true 80. The following are sign of right ventricular failure, except a. Rales b. Hepatomegaly c. Increase JVP d. Ascites dan oedems 81.

82. What is common associated risk factor for CAD a. Dislipidemia b. Sedentary of life c. Cigarette smoking d. All above are true 83. Imbalance O2 supply and demand to the miocardium IHD 84. Karakteristik dari infark miokard, kecuali a. Chest pain more than 15 minute b. At times be precipitated by exertion c. Chest pain relieve after taking rest d. In right coronary artery stenosis, epigastric discomfort and syncope is one of the most symptoms. 85. All the following statements can be a cause of ischemic heart disease, except a. Spasme b. Atherosclerosis c. Plaque ruptur d. High HDL level 86. Hemodinamik exam having a meaning to diagnose RHD a. Hemoglobin level b. Number RBC c. Number WBC d. LED 87. To determine etiology carditis (endocarditis and miocarditis) require inspection a. Hematology b. Clinical chemistry c. Urinalysis d. Blood culture 88. Which one is not including triple cardiac marker a. CK MB b. Mioglobin c. Troponin d. LDH 89. Marker RHD severity a. ASTO b. CPR c. BSR d. WBC 90. The signs of the cardiac tamponade are a. Chest pain increased of the CVP, heart sound decrease b. Hipotensi, HS decrease, fracture of the sternal bone c. JVP , HS decrease, hipotensi d. Fracture of the ribs, hipotensi, normal HS 91. 92. 8

93. 94. Aritmia mungkin disebabkan ketidaknormalan struktur jantung Mitral Stenosis 95. Karakteristik supraventrikuler tachycardi adalah ... 96. Fact about ventrikel aritmia a. Impulse originating from an area proximal to the AV junction b. Have preceding corresponding P wave and the QRS compleks c. Ventrikuler takikardi is a fast, irregular beating of the heart d. The extend of the ventrikuler takikardi determine the course and necessity of treatment. 97. The right position that used for cor PA, left lateral, LAO,RAO. 98. 99. Double contour sign of enlargement / hipertrofi a. Right ventrikel b. Left Atrium c. Right atrium d. Left ventrikel 100. Pemeriksaan jantung, kecuali a. Ventriculography b. Plain radiografi photo c. Heart Fluoroscopy d. Echocardiography 101. CTR : perbandingan cardio dan thorax 102. 103. Thoracix radiographic photo (PA) are done with a. Standing position b. Film on behind c. Supine position d. X-ray on front 104. Picture of enlargement of left vntrikel apex downward 105. 52 thn, , decrease visual acquify di kedua matanya. Hasil funduskopi : Internal bleeding, edema retina, hard exudately, vena dilatasi, dan berdarah. Diagnosisnya a. Central serous retinopathy b. Age related macular degeneration (ARMD) c. Retinal detachment d. Diabetic retinopathy 106. A 75 years old man complain blurred vission in his right eye. In fundus photograph showed surpra haemorrhages in right eye. The diagnosis a. Retinal detachmnet b. CRVO c. BRVO d. Hypertensive retinopathy 9

107. 108. Fundusphotograph pada CRAO 109. 110. Fundus fotografi ditemukan pada pasien dengan malignan hipertrofi a. Optic atrofi b. Optic sweller c. Retinal bleeding 111. True for mild and moderate hipertensi a. Retinal bleeding b. Arteriolar narrowing c. Retinal oedema d. Microaneurism 112. 113. Less sportif in RHD a. Carditis b. Migratory Poliarthritis c. NA d. Subacute nodule 114. 115. 116. The most common etiology of myocarditis a. Fungae b. Bacterial c. Viral d. Tropozoa 117. Myxoma located a. LV b. LA c. RV d. RA 118. The characteristic lesion of infective endocarditis is a. Inflammation b. Vegetation c. Destructive of valve d. Differentiation of valve 119. Acute pericarditis caused by as follows, except a. Microbacteria b. Neoplastic disease c. Degeneration d. Tropozoa

10

120. Infeksi dari mikrobakteri pada permukaan endothel jantung disebut a. Pericarditis b. Miocarditis c. Infeksi endokarditis d. RHD soal yang tercatat = 97 dari 120 soal. eNBe : 4m all 2 all 1. Sistem pencatatan, NO ABSEN & + 130. 2. Yang tidak mendapat bagian mencatat soal (soal kurang dari 130), catat inti-inti skenario. Misal jumlah soal 120, absen no 121 mencatat skenario pertama dst., 1 skenario boleh dicatat 2-3 orang. Kerja sama antara kalian sangat diharapkan. Kompak2lah y0w! 3. Yang udah tau info ini, kasih tau ke teman lain yang belum tau, key! 4. Pilihan gandanya (a,b,c,d,e) dicatat juga, disitulah qt bisa belajar lebih. Lagipula untuk tipe multiple choice question (soal pilihan ganda) pilihan jawabannya terkadang sedikit mengecoh. 5. Kalau bisa tetap dalam versi inggris, kadang ada yang sedikit salah mengartikan soal. Tulisannya juga!! g mesti bagus2 bener yang penting terbaca. 6. Seandainya tahu jawabannya langsung dijawab (klo bisa cari tahu jawabannya), tapi klo g yakin g dijawab juga gpp. SERSAN,serius tapi santai., Santai!! n_n thx 4 all

11