Professional Documents
Culture Documents
2012
PATIENTS IDENTITY
Name : Mr. M Age : 65 years Sex : Male Address : Bandorasakulon Cilimus Religion : Islam Nationality : Indonesian Admission Date : May 4th 2012 Room : Elisa RM : 136269 Diagnosis : Mild Head Injury
: (-) : (-)
GENERAL
EXAMINATION
General Appearance : Moderate Consciousness : Compos Mentis GCS : 15 Blood pressure : 160/100 mmHg Pulse : 80 x/minute Respiration : 20x/minute Temperature : 36,7 0C
PHYSICHAL
EXAMINATION
Skin : Anemis (-), Icteric (-), Cyanosis (-), normal turgor Head :Exscoriation 3 x 1 cm Battles sign (-) Eyes : Anemic Conjunctiva -/Icteric Sclera -/-, Pupil round, isokor, Light reflex +/+ reactive Raccoons eye -/-
CONT
Nose Neck
Chest
Cor
Pulmo
: No abnormalities epistaxis -/: No lymph node enlargement, mass (-) Meningeal Reflex : Shape and movement simetric in both sides : Nomal heart sound, reguler, shuffle (-) : VBS +/+, rales -/-, wheezing -/-
CONT
Abdomen Inspection Auscultation Palpation
Anus-Rectum : Normal Inguinal and Genital : not examined Extremities : Acral warm +/+
LABORATORY FINDINGS
May 4th 2012 Hb : 13,1 g/dl Ht : 38 % WBC : 11.400/mm3 RBC : 4,57 million/mm3 Trombocyt : 223.000/mm3 MCV : 84 fl MCH : 29 pg MCHC : 34%
CONT
Sodium : 144,5 mmol/dl Pottasium : 3,83 mmol/dl Chloride : 102,5 mmol/dl
CT SCAN IMAGING
TREATMENT
Inpatient O2 via nasal canule Intravenous fluid Ringer Lactate 1000cc/24 hour Metampiron and Diazepam 3 times a day Multivitamins 3 times a day p.o Beta histine 3 times a day p.o Ranitidine 3 times a day p.o Ketorolac twice a day p.o Citicholine 3 times a day p.o Metochlorpamide 3 times a day i.v Mannitol 125 cc 4 times i.v Peptisol 150 mg 6 times a day
PROGNOSE
Quo ad vitam Quo ad functionam Quo ad sanationam
THEORY
HEAD INJURY
DEFINITION Head injury is both directly and indirectly head mechanical trauma that cause neurological function disturbance that is include physical disturbance, cognitive and physcosocial function that temporaly and permanently.
EPIDEMIOLOGY
Head injuries are among the most common types of trauma seen in NOrth American emergency departments (EDs), with an estimated 1 million cases seen annually. Many patients with severe brain injuries die before reaching a hospital , and almost 90% of prehospital trauma-related deaths involve brain injury.
ETIOLOGY
EPIDURAL HEMATOMA
Epidural hematomas typically become biconvex in shape as they push the adherent dura away from the inner table of the skull. Massive hematome >> caused by A.meningea media or sinus venosus that perforate
SUBDURAL HEMATOMA
Subdural hematomas appear to conform to the contours of the brain. Subdural hematomas may grow to cover the entire surface of the hemisphere. Furthermore, the brain damage underlying an acute subdural hematoma is typically much more severe that with the epidural hematomas.
INTRACERECRAL HAEMORRHAGE
Is brain parenchim bleeding, caused by intracerebral arterial rupture In period of hours or days, evolve to form an intracerebral hematoma or a coalescent contusion with enough mass effect to require immediately surgical evaluation For this reason, patients with contusions should undergo repeat CT scanning to evaluate for changes in the pattern of contusion 12 to 24 hours after the initial scanerebral arterial leakage
Minimal intracerebral haemmorhage at left frontal regio Subdural higroma at right frontotemporal regio
Theory
Inpatient O2 via nasal canule Intravenous fluid Ringer Lactate 1000cc/24 hour Metampiron and Diazepam 3 times a day Multivitamins 3 times a day p.o Beta histine 3 times a day p.o Ranitidine 3 times a day p.o Ketorolac twice a day p.o Citicholine 3 times a day p.o Metochlorpamide 3 times a day i.v Mannitol 125 cc 4 times i.v Peptisol 150 mg 6 times a day
THANK YOU