Professional Documents
Culture Documents
Shiver
single, solemn shiver (with a maximum
duration of 1/2h) in lobar pneumonia;
repeated and intense shiver (tuberculosis,
abcesses, pleuritis).
Sweat
all feverish pulmonary diseases;
during night time in tuberculosis.
Anorexia and loss of weight
tuberculosis
broncho-pulmonary cancer
bronchopulmonary suppuration.
Local symptoms
Chest pain
It is a subjective manifestation;
The pain may be connected with the thorachic
wall, the vertebral column, the pleura, the heart and
the large vessels, esophagus, mediastinum or with
abdominal organs.
It may be felt as a diffuse, violent pressing, burn or
constriction.
1.Parietal causes
-Zona zoster: vesicular eruption on the intercostal
nerve tract, surrounded by a red erythema, preceded
and followed by intense pain
-Dermatomiositis: polymorf eruption associated with
a rough, painful edema of the muscles in that area.
- Cellulitis: inflammation of the adipose tissue,
accompanied by edema.
- Trichinosis: helminthiasis characterized by fever,
eosinophilia and thoracic pain; the final diagnosis is
made by muscle biopsy.
- Thoracic trauma: rib fractures chest pain associated
with polypneea and bone crepitations.
- Diseases of the mammary gland: mastitis, breasts
cysts, breast cancer.
- Osteitis and periostitis (lues, tuberculosis).
- Intercostal neuralgia : pain in Valleix points (located
along the intercostal area paravertebral, axillar,
parasternal).
- Tietze syndrome : painful tumefaction of the condro-
costal joints of the ribs 1 and 2, and more rarely ribs 3
and 4; usually the disease is unilateral.
2. Vertebro-medular causes
- Vertebral tuberculosis (Potts disease)
- Spine cancer (progressive pain), osteomyelitis;
spondylosis
- Hernia of an intervertebral disc (compression of the
nerve in the conjugation hole)
- Tabes (chronic luetical meningitis involving the
posterior root of the spinal nerves with painful crisis)
3. Respiratory causes
Characteristics of the pain
- felt like a dagger in the thorax
- stronger during respiratory movements,
coughing and pressing
- on the involved side, the respiratory
movements are superficial and the patient will lay
down on the opposite side.
Causes
- Dry pleurisy or pleuritis: lancinating, sharp, unilateral
pleuritic pain which is felt over the involved area. The
patient will lay down on the opposite side in order to
avoid pressure on the involved side. The pleuritic pain
disappears when the pleural effusion appears.
- Pleural effusion: the patient will lay down on the
involved side to permit the uninvolved side to have an
more amplified inhaling.
- Purulent pleurisy: violent pleuritic pain and a very
sensitive hemithorax. The patient will lay down on the
opposite side.
- Diaphragmatic pleurisy: very violent pleuritic pain,
sometimes unbearable, located at the diaphragmatic-
costal insertion and affecting the intercostal and the
phrenic nerve; sometimes the pain can radiate in the
abdomen;
phrenic neuralgia is characterized by hyper
sensitivity when pressing the following areas:
- scalen spot (between the 2
sternocleidomastoidian ends)
- parasternal points
- diaphragmatic button (at the junction
of the middle of the breast bone with the extension of the
- Interlobal pleurisy: chest pain along the lung cleavage
(scarf pain)
- Pneumothorax: very violent, dagger-like sensation,
associated with anxiety, cyanosis, dyspneea, weak and
frequent pulse, diminished vesicular sounds and
characteristic modifications on the chest x ray.
- Lobal pneumonia: localised in the nipple region, lasting 2-
3 days and then disappearing;
- Pulmonary embolism: strong, violent pain, like a dagger,
dyspneea, cough, cyanosis, tachycardia, lypothymia and
syncope.
- Pulmonary cancer: progressive pain, which in advanced
stages becomes unbearable, doesnt respond to common
medication; the site depends on the size and location of the
-Pancoast Tobias syndrome (syndrome of the
pulmonary peak):
malignant tumor of the lung apex which
infiltrates the nervous plexues and the large vessels.
asthenia, anorexia, subfeverishness, dry cough,
hemoptysis, cyanosis
it can be associated with Claude Bernard-
Horner Syndrome (enophtalmia, miosis resulting from
cervical sympathic compression).
4. Cardiovascular causes
- Pain usually appear in the precordial and
retrosternal area: chronic ischemic cardiopathy,
exertional and decubitus angina pectoris, myocardial
infarction, dissecting aortic aneurysms, pericarditis.
.
6. Abdominal causes
Phrenic abscess; inflammation of the colon
flectures; billiary lithiasis, spleen diseases.
7. Mediastinal causes
Mediastinitis, mediastinal tumors are associated
with cyanosis, rain coat edema, collateral circulation.
Dyspneea
Definition
difficult breathing
the main symptom of a respiratory insufficiency
its manifestations are objective and subjective
subjective: thirst for air and objective: forced
respiratory movements
Classification
a)Depending on frequency
- polypneea (tachypneea) an increase in the
respiratory frequency per minute
- bradypneea: a decrease in the respiratory
frequency per minute
Normal frequency: 16-18/min.
b) Depending on amplitude:
- hyperpneea: an increase in the breathing
amplitude
- hypopneea: a decrease in the breathing
amplitude
c) Depending on the phase of respiration
- inhaling dyspneea
- exhaling dyspneea
- mixted dyspneea
d)Modifications in the respiratory rhythm:
Cheyne-Stokes breathing
Kussmaul breathing
Biot breathing
e) Depending on conditions of appearance
- exertional dyspneea
- rest dyspneea
f) Depending on timing and onset
- paroxysmal dyspneea (bronchic asthmatic crisis,
cardiac asthma, acute pulmonary edema, left heart failure)
- vesperal dyspneea (it appears in the evening left
ventricular failure)
- acute dyspneea (upper respiratory ways
obstruction, acute pneumopathies, pneumothorax,
pulmonary infarction, acidosis)
- dyspneea with a slow evolution (chronic
inflammation of the respiratory ways, pulmonary tumors,
pulmonary emphysema)
Accelerating rhythm dyspneea -Polypneea
(tachypneea)
- physiological (emotions, physical exercise)
- pathological
a) feverish conditions when the high
temperature of the circulating blood excites the respiratory
centers
b) diseases which lead to a decrease in the
amplitude of respiratory movement (intercostal neuralgies,
costal fractures, cifoscoliosis, respiratory muscles
paralysis)
c) diseases which lead to a decrease of the
lungs breathing area (pulmonary embolism, pulmonary
edema, pleural effusions, pneumonia, pulmonary tumors)
d) large bleedings, CO intoxication
e) diseases of the cardio-vascular system
f) intraabdominal processes that limit the
diaphragmatic excursions (ascites, tumors)
Thining out rhythm dyspneea- Bradypneea
It may be:- inhaling, exhaling, mixed .
a)Inhaling bradypnoea: decrease in the upper respiratory
ways calibre, leading to obstructive ventilatory disturbances.
Causes
- internal obstacles: the vocal cordes paresis, croup,
pseudocroup, glotic oedema, laryngeal tumours.
- external compression: neighbouring tumours,
mediastinal tumours
Accessory inhaling muscles are involved and this leads to
an ortopneea and a prolonged and deep inhaling
accompanied by a sound that is called pathological wheezing.
In severe obstruction, in spite of the effort, only a small
quantity of air comes in, therefore the inter-alveolar pressure
is lower than the atmospheric pressure, phenomenon that
leads to the retraction of the tissue parts during inhaling; this
is called draught.
b) Exhaling bradypneea - an obstacle stops the evacuation
of the intrapulmonary air, creating a whistling sound
called wheezing.
Causes : - bronchic asthma in crisis
- chronic obstructive pulmonary disease (chronic
bronchitis and pulmonary emphysema).
Typical example: asthma in crisis- sudden onset,
frequently at night, suffocating sensation, prolonged
exhaling, ortopneea, associated with anxiety, cyanosis.
Initially, dry cough, then mucous sputum, pearl-like
aspect.
The mechanism of bronchic obstruction in asthma: edema,
bronchospasm, hypersecretion.
Expectoration
- consists of the oral elimination of pathologic secretions
accumulated in the respiratory ways, during the coughing
process
- the product of expectoration is the sputum
Sputum results from:
mucous and serous gland secretions which
develop in inflammation and bronchic asthma.
exudation :inflammation of the aveolas and of the
bronchi which appears in pneumonia and broncho-pneumonia.
transudation which appears in acute pulmonary
edema and in congestive heart failure
blood resulting from pulmonary distruction
foreign bodies
saliva
nasal pharyngeal secretions
pathological collections in the pleura, mediastinum
or subdiaphragmatic collections which open in the respiratory
ways (vomica)
The sputum has a great diagnostical value:
Rusty, brownish sputum appears in lobal pneumonia.
Cranberry jelly - like sputum appears in the
pulmonary cancer.
Rich, purullent sputum appears in bronchiectasia.
Fetid sputum appears in pulmonary abscess and
gangrene.
Macroscopically we describe the quantiy, aspect, colour, smell.
a) The quantity depends on the nature of the pathological
process, the evolution of the disease, the intensity of the cough
and the efficiency of the treatment.
- rich sputum :bronchiectasia, pulmonary
tuberculosis, abscess, pulmonary gangrene, acute pulmonary
edema, purulent pleurisy, hydatic cyst which opens in the
respiratory ways
- reduced sputum: early stages of acute
tracheobronchitis, early stage of tuberculosis, bronchic asthma
Vomica
represents the sudden and rich expectoration
resulting from the opening in the airways of a collection
situated in the pulmonary parenchyma (pulmonary abcess,
cavitary tuberculosis, hydatic cyst), pleura (purulent
pleurisy) or the neighbouring organs (subphrenic or
mediastinal abcess).
it is preceded and accompanied by coughing
it can be massive, medium and low; unique or
fractionary; purulent or spring water like.
Massive vomica appears in large pulmonary abcesses,
purulent pleurisy, hydatic cyst and is associated with
cyanosis, asphyxia sensation, anxious facies.
b)The aspect
- mucous sputum: viscous, adherent, well aired, formed
from the mucine of the secreating glands.
appears in: acute bronchitis, bronchic
asthma, pneumonia, early pulmonary tuberculosis; in bronchic
asthma, at the end of the crisis it presents small, opalescent
sediments, called pearled expectoration , accompanied by
spiral filaments called Curschmanns spirals.
- muco-purulent sputum: yellowish or greenish blend of
mucus and pus. It appears in: acute bronchitis, pneumonia,
broncho-pneumonia, bronchiectasia, cavitary pulmonary
tuberculosis.
- purulent sputum: yellow-greenish cream - like sputum.
It appears in chronic bronchitis, bronchiectasia, pulmonary
abcess, cavitary pulmonary tuberculosis. In case of vomica,
this will consist of pus.
- serous sputum: transparent, white - pink foaming
sputum. It appears in pulmonary edema, left ventricular
- sero-muco-purulent sputum: a blend of serosity, pus
and mucus. It appears in fetid bronchitis, pulmonary gangrene
and is characteristic for bronchiectasia. This sputum has four
layers: foaming, serous, muco-purulent and purulent (rough-
grained).
- pseudo-membrane like sputum: liquid part in which
solid bodies float; very rich in fibrine that coagulates and
forms pseudo membranes: diphteric croup, pseudo membrane
like chronic bronchitis
- sanguinolent sputum: blend of mucous, serous,
purulent secretions in which variable quantities of blood
appear. It appears in pneumonia, acute pulmonary edema,
bronchopulmonary cancer, pulmonary gangrene.
c) The colour:
white (mucous secretion), yellow-greenish (purulent
secretions), pinky (serous secretion), reddish (hemoptoic
secretions), rusty, brownish sputum (lobal pneumonia)
- red, adherent sputum (pulmonary infarction)
- cranberry jelly - like sputum (broncho-pulmonary
cancer)
- brownish sputum (pulmonary gangrene)
- black sputum in people working in coal mines.
d) The smell
It does not usually smell.
The sputum can have a fetid smell in bronchiectasia,
pulmonary abcess, pulmonary gangrene.
Hemoptysis
elimination of blood at the level of the respiratory system.
mechanism: breakage or ulceration of a vessel;
blood diapedesis at the surface of the pulmonary
alveolas or in the bronchi.
the differential diagnosis is made with posterior epistaxis,
stomatorahia, hematemesis.
the blood in hemoptysis is red, rutilant, aired, partially
uncoagulable, covered by a thick layer of sputum. This appears
in patients who have suffered of respiratory diseases before.
the blood in hematemesis comes from a patient suffering
from a digestive disease, is coagulated and unaired
hemoptysis is accompanied by a retrosternal warming
sensation, diziness, respiratory uneasiness, cough.
the patient is pale, anxious, experiencing cold sweat,
dyspneea, tachycardia, lipothymia.
large hemoptysis are lethal by asphyxia and shock.
Causes:
- Respiratory diseases: pulmonary tuberculosis, broncho-
pulmonary cancer, bronchiectasia (repeated haemoptysis),
pulmonary abcess and gangrene, acute tracheo-bronchitis (it
appears in violent cough), thoracic traumas, hydatic cyst,
pulmonary infarction, traumas.
Nails
Clubbing fingers are present in the following respiratory
diseases:
pulmonary tuberculosis
broncho pulmonary cancer
bronchiectasia
pulmonary supuration, pulmonary sclerosis
empyema
The Subcutaneous Cellular Tissue
generalised edema (anasarca) appears in cardiopathy,
nephrotic syndromes; liquid is present also in the pleural
cavity - hydrothorax.
raincoat edema appears in the obstructions of the superior
vena cava by an expanding mediastinal process
Quincke edema and bronchic asthma are both allergic
processes in which the target organ is different.
In Quincke edema the target is on the tegumentary and
subcutaneous cellular tissue, while in bronchic asthma it is at
the bronchiole level.
This is why there appear allergic symptoms.
The Physical Examination of the Upper Respiratory Ways
The nostrils:
The most important thing to be observed is the nasal
obstruction. The patients will breath orally, this being a
very important aspect, because the air is warmed and
purified in the nose.
The nasal voice:
In order to determine the obstruction examiners ask the
patient to breathe nasally while pressing alternatively
the nostrils. The nature of the obstacle will be
determined with the nasal speculum.
The sinuses:
They can be the source of an inflammation which may
result in a frontal headache. It is termed sinusitis. If
vivid sensitive reaction results when pressure is applied,
acute sinusitis is suggested.
The Physical Examination of the Trachea
1.INSPECTION
Teguments for colour, scars of previous heart or lung
surgery; swellings, marks and spots on the skin (eruptions,
collateral circulation, edema)
Thorax conformation (normal, deformed)
Respiratory type
The frequency and amplitude of the respiratory movements
Modification of the thorax during respiration
-
Thoracic teguments examination
- vesicles in the intercostal area suggests zona
zoster.
- tiny vein dilatation on the top of the thorax in
pulmonary peaks tuberculosis.
- located edema with a bucket appears in deep
suppurations (empyema).
Thorax conformation
Normal:
the xiphoid angle is of ninety degrees
the antero-posterior diameter is smaller than the
transversal one.
the vertebral column has a very slight curve with an
anterior concavity (physiological ciphosis).
the supraclavicular and subclavicular fossi are not
encaved nor protruding.
the ribs are bent anteriorlly and downwards.
Thoracic deformations:
bilateral (symmetrical and asymmetrical)
unilateral.
a) Bilateral Symmetrical Deformations
The emphysematous thorax: The thorax is dilated and rigid
like a barrel (always while inhaling) with an antero-
posterior diameter bigger or equal to the transversal one;
The ribs are horizontal, the supra and subclavicular areas
protrude; Xyphoid angle is larger than ninety degrees, and
the respiratory movements are very weak.
The paralytic thorax: The antero-posterior diameter is smaller
than the transversal one; The xyphoid angle is under ninety
degrees, the supra/subclavicular areas are deepened. Most
of the cases are congenital, but it can be secondary to
tuberculosis.
The conoid thorax: The lower part of the thorax is very dilated
due to a hepato or splenomegaly or an ascitis.
The adenopatic thorax: The ganglion growth results in the
dilatation of the upper part of the thorax..
Pectus excavatum (funnel chest) = localized depression of the
lower end of the sternum or depression of the whole length of
the sternum.
c) Unilateral Deformations
-Retraction: pachypleuritis, massive atelectasis (the lack of air
in the thorax), extensive pulmonary fibrosis.
Below this level is dullness from the liver. The patient is then
instructed to exhale as much as possible, and the percussion
is repeated.
With expiration, the lung contracts, the liver moves up, and
the same area becomes dull; that is, the level of dullness
moves upward.
Pathological situations:
a) the lowering of the inferior edge of the pulmonary
resonance:
pulmonary emphysema (alveolary distension)
bronchic asthma during crises (hyperinflation)
bronchiolitis
b) the ascending of the inferior edge of the pulmonary
resonance:
- unilateral: pleurisy, hepathomegaly, splenomegaly,
diaphragmatic hernia.
- bilateral: ascitis, severe meteorism, advanced
pregnancy
c) the diminished or abolished active mobility
- bilateral: pulmonary emphysema, generalised
peritonitis.
- unilateral: pleural effusion (exudate), pahipleuritis,
tumours
Dulness appears in:
- modifications in the thoracic wall- obesity, tumours
- the interposition of a non-containing air medium
between the lung and the thoracic wall
- pulmonary parenchyma without air (pneumonia,
pulmonary infarction, tuberculosis, atelectasis, pulmonary
cancer, pulmonary abcess, hydatic cyst).
Pleural diseases with dullness
a) The exudative pleurisy
- dullness depends on the quantity of pleural liquid
- when effusion exceeds 200 ml, we have subdullness
combined with dulness in the inferior pulmonary region
- in moderate pleurisy, the superior limit of the dullness
draws a curved line called the Damoiseau line
- when the liquid is abundant, the superior edge is a
horizontal line
- the pleural effusion compresses the neighbouring parenchyma,
resulting in its relaxation in the pleurisy proximity area; the
result of the percussion is called skodism (tympanic sound)
- the massive pleural effusions displace the adiacent organs (the
heart, the mediastinum)
- if the healing process is defective, the pleural layers get thick,
leading to persistent dulness or subdullness
b) Hydrothorax (transudate)
-Appears in heart failure, renal syndromes (nephrotic)
-The presence of a small quantity cannot be proven by
percussion
-Usually bilateral
Differential between pleurisy and hydrothorax
in pleurisy the superior limit of dullness is not changed
by the patient position because of the adherences
the adiacent organs are not dislocated in hydrothorax if
bilateral
c) Hemothorax the presence of blood in the pleura (thoracic
trauma, costal fractures, pleural puncture, aorta aneurysm
broken in the pleura, bleeding disorders)
d) Pneumo or pio pneumothorax the presence of air in the
pleura
- the air entrance is
acompanied by germs which leads to inflammation, with the
appearance of a pleural effusion; suprainfection leads to
piopneumothorax
e) Pleural tumours (benignant or malignant)
They influence dullness on a limited region
At this level, the vocal fremitus is abolished and a respiratory
immobility of the affected hemithorax is experienced
In malignant tumours, a pleural effusion appears.
f) Pleural thickening, pahipleuritis
- They determine costal and mediastinal retractions, scoliosis
abolished vocal fremitus, dullness, respiratory silence.
Pulmonary diseases with dullness
a) Infiltrating processes lobal pneumonia, bronchopneumonia
pulmonary tuberculosis, pulmonary infarction, pulmonary
tumours.
Pneumonia
-Very abundant exudation that fills the involved alveolas and
generates dullness + augmentative vocal fremitus
- The pneumonic lobe generates compression on the
neighbouring lobe, which will be relaxed, determining skodism
- The dullness is not as resistant as in pleurisy
Pulmonary tuberculosis
- Over 50% of cases are located apically and subapically
Pulmonary infarction
-It generates dullness if the infarcted area is superficial
Pulmonary tumours
-A tumour is a compact tissue without air
- They generate variable dullness or subdullness with great
Pulmonary cavities
(tubeculose cavities, abcess, pulmonary gangrene, hydatic cyst)